By Ramzaj
Councils about care of a problem skin. How to choose cosmetics for skin purification at acnes.
The correct care of a skin it is more narrow half of victory over spots and acnes. The care of a skin will allow to intercept new rashes, to reduce treatment terms, will reduce requirement for medicines and cosmetic procedures.
1. To begin with try not to squeeze out spots. The occluded comedoes and inflammatory devices of an eruption cannot neither be squeezed out, nor to dissect. It follow-up damages a skin, enhances an inflammation, promotes an infection contamination and can cause scars. If you cannot overcome desire that revert to the expert.
2. Wash the face twice a day, but be not overzealous with washing, spots do not arise from a dirty skin. Too frequent washing can provoke an inflammation. Instead of ordinary soap it is better to use special, and also refining skins or gels (routinely names contain words cleansing, cleanser or directly specify in use at fat / to a problem skin, predilection to acnes). Wash simply arms, easy locomotions, with warm water, after washing put a towel, instead of triturate it a skin. Yield a skin to dry up 10-15 minutes before drawing of any agents against spots.
3. If at you "oily" type of a hair, wash them every day with suitable shampoo. Do not allow a hair to occlude a face skin, forehead, temples. Avoid cosmetics fat and containing an oil for a hair.
4. Use such cosmetics and agents of hygiene which suit a fat skin. Normally they contain an inscription «non-comedogenic», i.e. do not promote formation of comedoes, an occlusion of dermal pores. Same concerns a make-up and protective sunburn creams. At use of many aboriginal drugs from spots wetting agents (moisturizer, it is ordinary this milk) will be necessary for you. They can possess sun-protection properties (SPF).
5. Spare a skin from an abudant solar irradiating. Do not use sun decks. That the sun itself will cure spots - a myth, instead of a reality. Sunburn only masks spots. Tendency promptly to receive sunburn quite often conducts to skin photodamage, and it threatens with a carcinoma cutaneum, a melanoma. Some agents of treatment of acnes increase sensitivity of a skin to an ultraviolet.
6. Avoid abrasion of a skin in those places where routinely there are spots. This council concerns not only clothes and dressings, but also the playing sports bound to use of protective inventory (top sections, overlays, etc.). The open skin should not adjoin to them.
7. For daily care of a problem face skin, inclined to acnes, it is released much only. For example, the scraps, helping to peel high layer of a skin and to discover pores (it is possible to use 1-2 times a week if there is no inflammation). Try also refining lotions and napkins - 2 times a day. Choose soft, not the revellents approaching your skin. Some agents on a leaving can not be combined with peeling drugs for treatment of acnes.
If you already apply any outside agent to treatment of acnes, yield it to work. Skin purification make not after, and before drawing of a medical drug. Choose for all it suitable time.
The correct care of a skin will bring to you a lot of advantage. But do not count that only care of a skin will relieve you of spots. Such it is possible not always and not to all.
© Ramzaj
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Monday, May 21, 2007
Sunday, May 20, 2007
Hormones And Skin
By Ramzaj
Man's sexual hormones and skin. Testosterone, other androgens also that on them influences.
Formation of dermal grease is boosted by man's sexual hormones - androgens. The most active androgens - testosterone, 5-testosterone (dihydrotestosterone, DHT) and 5-androstendiol. Despite of the name, man's sexual hormones is and at women. Androgens are produced by sexual glands: at men in testicles, at women - in ovaries. Both female, and man's sexual glands are checked by hormones of a pituitary body - a member which is being a brain.
testosterone
dihydrotestosterone (DHT)
5-androstendiol
In a skin activity of androgens strengthens some ferments (an androgen - reductases I of type, 5-alpha-reductase). These ferments transmute less active androgens into more active: testosterone and DHT (5-testosterone). Crates of a follicle have receptors of androgens and can accept signals from them.
Changes in a level of the hormones, descending during puberty, explain occurrence of spots in teenagers.
Activity of sebaceous glands can variate and at some diseases of endocrine system: a pituitary body, paranephroses, or man's and female sexual glands. The quantity of dermal grease on a skin thus increases or contracts.
The reinforced formation of dermal grease, seborrhea and acnes can educe not only from pinch of the common level of sexual hormones in a blood. The most part of testosterone in a blood is bound by special protein SHBG (the globulin binding sexual hormones). Deficiency SHBG result ins to strengthening of the free testosterone which is joined to receptors in sebaceous glands. Activity of ferment 5-alpha-reductases can be increased at some people. As a result at these people on sebaceous glands react more active androgens. Sensitivity of a skin and sebaceous glands to androgens at some people also appears higher.
Female sexual hormones - the estrogens used in hormonal contraception, reduce formation of dermal grease. The same effect drugs - can yield antiandrogens.
Purpose of drugs of testosterone to boys up to period of puberty (or progesterones with androgenic properties) boosts formation of dermal grease ice and promotes development of acnes.
And still, androgens - not the single hormones influencing operation of sebaceous glands. Similar influence is rendered with a growth hormone and an insulinoid growth factor.
Some states related to the hormonal status of the person, can worsen a state of a skin. It is a syndrome of polycystic ovaries (at women), Cushing syndrome (from the long-term treatment by steroid hormones). Also failure of ferments which control formation of sexual hormones: for example, sterol-hydroxylases, that descends very seldom, and also depression and the strong psychologic stress.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Man's sexual hormones and skin. Testosterone, other androgens also that on them influences.
Formation of dermal grease is boosted by man's sexual hormones - androgens. The most active androgens - testosterone, 5-testosterone (dihydrotestosterone, DHT) and 5-androstendiol. Despite of the name, man's sexual hormones is and at women. Androgens are produced by sexual glands: at men in testicles, at women - in ovaries. Both female, and man's sexual glands are checked by hormones of a pituitary body - a member which is being a brain.
testosterone
dihydrotestosterone (DHT)
5-androstendiol
In a skin activity of androgens strengthens some ferments (an androgen - reductases I of type, 5-alpha-reductase). These ferments transmute less active androgens into more active: testosterone and DHT (5-testosterone). Crates of a follicle have receptors of androgens and can accept signals from them.
Changes in a level of the hormones, descending during puberty, explain occurrence of spots in teenagers.
Activity of sebaceous glands can variate and at some diseases of endocrine system: a pituitary body, paranephroses, or man's and female sexual glands. The quantity of dermal grease on a skin thus increases or contracts.
The reinforced formation of dermal grease, seborrhea and acnes can educe not only from pinch of the common level of sexual hormones in a blood. The most part of testosterone in a blood is bound by special protein SHBG (the globulin binding sexual hormones). Deficiency SHBG result ins to strengthening of the free testosterone which is joined to receptors in sebaceous glands. Activity of ferment 5-alpha-reductases can be increased at some people. As a result at these people on sebaceous glands react more active androgens. Sensitivity of a skin and sebaceous glands to androgens at some people also appears higher.
Female sexual hormones - the estrogens used in hormonal contraception, reduce formation of dermal grease. The same effect drugs - can yield antiandrogens.
Purpose of drugs of testosterone to boys up to period of puberty (or progesterones with androgenic properties) boosts formation of dermal grease ice and promotes development of acnes.
And still, androgens - not the single hormones influencing operation of sebaceous glands. Similar influence is rendered with a growth hormone and an insulinoid growth factor.
Some states related to the hormonal status of the person, can worsen a state of a skin. It is a syndrome of polycystic ovaries (at women), Cushing syndrome (from the long-term treatment by steroid hormones). Also failure of ferments which control formation of sexual hormones: for example, sterol-hydroxylases, that descends very seldom, and also depression and the strong psychologic stress.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Monday, May 14, 2007
Why There Are Scars?
By Ramzaj
Scars after acnes as a consequence of an inflammation and fracture of tissues. Phylums of scars after pimples.
Scars are formed in those places where there is the strong damage and fracture of tissues. In a skin it is the copulative tissue surrounding hair follicles, and sometimes - hypodermic cellulose.
Scars arise during response of tissues to damage, process of an adhesion of wounds. When something collapses, formed "hole" is closed up by those materials of which the copulative tissue is constructed. First of all, it is a collagen. Process of an adhesion and a cicatrization of each damage lasts about one month.
"Repair" of tissues descends in terminating stage of an inflammation. At an inflammation of a tissue are always damaged. When it is enough fracture greater, or process of an adhesion is broken, the former structure of a tissue sometimes is not recovered. The scar is formed on its place.
At acnes are formed two phylums of scars - atrophic and hypertrophic. The arrest of development means a deficiency of propagation, and a hypertrophy - excess. The strong or feeble ability of a copulative tissue form a collagen can to be a specific feature of the person. On different fields of a skin formation of a cicatrical tissue goes with different intensity, it depends on blood supply of the yielded area. For example, on cheeks occurrence of scars more possibly, than on temples.
At atrophic scars formation of a collagen goes with small intensity, it suffices only on repair of walls of "hole". Such holes are the flaws of the skin which have arisen after fracture of a follicle and a copulative tissue surrounding it, and even hypodermic cellulose. Fracture can be greater - and scars can be deep and wide. It is characteristic for heavy shapes of disease.
Besides attempts to squeeze out and dissect spots invoke additional damage of tissues - so, and occurrence of scars. Thus in a wound the infection contamination that else complicates an adhesion can be brought.
At hypertrophic scars formation of a collagen goes more strongly, than it is required, and the scar rises above a surface of a skin. The expressed hypertrophic scars-keloids it is possible to expect at the people predisposed to such response at a genetical level. They have inborn features of synthesis of a collagen and formation of a scar goes more longly - for example, at many people with a dark skin.
Most the poor in scars - that they do not peter. They remain for a long time, and without treatment for ever. Scars cannot dissolve. Any treatment of scars includes their physical removal - bodily or stratums.
During an aging people lose a dermal collagen, approximately on 1 % a year, since 40 years. Therefore at older persons some scars become more appreciable, and others - are less appreciable.
The main thing that it is necessary to remember about scars are that at acnes they arise only as a consequence of an inflammation. The one who can prevent the strong inflammation, can avoid and occurrences of scars.
The main thing to make it, yet late.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Scars after acnes as a consequence of an inflammation and fracture of tissues. Phylums of scars after pimples.
Scars are formed in those places where there is the strong damage and fracture of tissues. In a skin it is the copulative tissue surrounding hair follicles, and sometimes - hypodermic cellulose.
Scars arise during response of tissues to damage, process of an adhesion of wounds. When something collapses, formed "hole" is closed up by those materials of which the copulative tissue is constructed. First of all, it is a collagen. Process of an adhesion and a cicatrization of each damage lasts about one month.
"Repair" of tissues descends in terminating stage of an inflammation. At an inflammation of a tissue are always damaged. When it is enough fracture greater, or process of an adhesion is broken, the former structure of a tissue sometimes is not recovered. The scar is formed on its place.
At acnes are formed two phylums of scars - atrophic and hypertrophic. The arrest of development means a deficiency of propagation, and a hypertrophy - excess. The strong or feeble ability of a copulative tissue form a collagen can to be a specific feature of the person. On different fields of a skin formation of a cicatrical tissue goes with different intensity, it depends on blood supply of the yielded area. For example, on cheeks occurrence of scars more possibly, than on temples.
At atrophic scars formation of a collagen goes with small intensity, it suffices only on repair of walls of "hole". Such holes are the flaws of the skin which have arisen after fracture of a follicle and a copulative tissue surrounding it, and even hypodermic cellulose. Fracture can be greater - and scars can be deep and wide. It is characteristic for heavy shapes of disease.
Besides attempts to squeeze out and dissect spots invoke additional damage of tissues - so, and occurrence of scars. Thus in a wound the infection contamination that else complicates an adhesion can be brought.
At hypertrophic scars formation of a collagen goes more strongly, than it is required, and the scar rises above a surface of a skin. The expressed hypertrophic scars-keloids it is possible to expect at the people predisposed to such response at a genetical level. They have inborn features of synthesis of a collagen and formation of a scar goes more longly - for example, at many people with a dark skin.
Most the poor in scars - that they do not peter. They remain for a long time, and without treatment for ever. Scars cannot dissolve. Any treatment of scars includes their physical removal - bodily or stratums.
During an aging people lose a dermal collagen, approximately on 1 % a year, since 40 years. Therefore at older persons some scars become more appreciable, and others - are less appreciable.
The main thing that it is necessary to remember about scars are that at acnes they arise only as a consequence of an inflammation. The one who can prevent the strong inflammation, can avoid and occurrences of scars.
The main thing to make it, yet late.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Saturday, May 12, 2007
Dermal Grease
By Ramzaj
Dermal grease: its composition and changes in different periods of life. Lipids and a protective mantle of a leather.
What consists of dermal grease? It is the complex intermixture from different materials similar to grease - lipids. The composition of dermal grease includes glycerides (more than 40 %), the free fatty acids (16 %), Aethers of wax (up to 25 %), squalene (12 %) and a cholesterin. Triglycerides of dermal grease mold up to the free fatty acids bacteria. Some of them form the volatile fatty acids, attaching has begun to smell to a leather. Dermal grease has no of odour.
On a surface of a leather grease is a part of a so-called protective mantle - a stratum formed by a secret of glands of a leather, its microflora and exfoliated corny scales. Because of acid medium on a surface of a leather (pH 4-5,5) it name also «an acidic mantle».
Dermal grease carries out some functions. It interferes with leakage of water and insiccation of a leather, protects from a fault acidic or an alkaline condition, interferes with a heading of bacteria. A mantle - the first protective barrier of a leather solving its many problems. Dermal grease is a natural care of a leather without which she would become dry, inclined to faults and flaws.
One of fatty acids of dermal grease, linoleic acid kills activity of the ferments participating during a keratinization of crates of a leather.
Dermal glands start to work for a nucleus, under activity of parent and natural hormones. The child is environed by caseous or waxy lubrication (lat. vernix caseosa). In the first 3-6 months after a birth a composition of dermal grease at the baby almost same, as well as at the adult. Then, up to 8-years age, in its composition becomes less wax and squalene, but it is more than cholesterin. During puberty production of dermal grease goes much more actively, being incremented almost to 5 times at men. At women of grease it is produced a little bit less, more appreciable decrease descends with the years - especially during a menopause.
Some diseases and the states related with endocrine systemic and hormones, can result in to pinch or decrease of production of dermal grease. The same effect is yielded also with some medicinal preparations.
Changes of a composition of dermal grease also can contribute to evolution of acnes - in particular, a deficiency of a linoleic acid. The increased content of grease result ins to change of a composition of lipids on a surface of a leather, to decrease of quantity of a linoleic acid - that can promote the reinforced keratinization in a follicle and its blockage.
The state at which production of grease and greasiness of a leather is appreciablly incremented, is called as a seborrhea.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Dermal grease: its composition and changes in different periods of life. Lipids and a protective mantle of a leather.
What consists of dermal grease? It is the complex intermixture from different materials similar to grease - lipids. The composition of dermal grease includes glycerides (more than 40 %), the free fatty acids (16 %), Aethers of wax (up to 25 %), squalene (12 %) and a cholesterin. Triglycerides of dermal grease mold up to the free fatty acids bacteria. Some of them form the volatile fatty acids, attaching has begun to smell to a leather. Dermal grease has no of odour.
On a surface of a leather grease is a part of a so-called protective mantle - a stratum formed by a secret of glands of a leather, its microflora and exfoliated corny scales. Because of acid medium on a surface of a leather (pH 4-5,5) it name also «an acidic mantle».
Dermal grease carries out some functions. It interferes with leakage of water and insiccation of a leather, protects from a fault acidic or an alkaline condition, interferes with a heading of bacteria. A mantle - the first protective barrier of a leather solving its many problems. Dermal grease is a natural care of a leather without which she would become dry, inclined to faults and flaws.
One of fatty acids of dermal grease, linoleic acid kills activity of the ferments participating during a keratinization of crates of a leather.
Dermal glands start to work for a nucleus, under activity of parent and natural hormones. The child is environed by caseous or waxy lubrication (lat. vernix caseosa). In the first 3-6 months after a birth a composition of dermal grease at the baby almost same, as well as at the adult. Then, up to 8-years age, in its composition becomes less wax and squalene, but it is more than cholesterin. During puberty production of dermal grease goes much more actively, being incremented almost to 5 times at men. At women of grease it is produced a little bit less, more appreciable decrease descends with the years - especially during a menopause.
Some diseases and the states related with endocrine systemic and hormones, can result in to pinch or decrease of production of dermal grease. The same effect is yielded also with some medicinal preparations.
Changes of a composition of dermal grease also can contribute to evolution of acnes - in particular, a deficiency of a linoleic acid. The increased content of grease result ins to change of a composition of lipids on a surface of a leather, to decrease of quantity of a linoleic acid - that can promote the reinforced keratinization in a follicle and its blockage.
The state at which production of grease and greasiness of a leather is appreciablly incremented, is called as a seborrhea.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Thursday, May 10, 2007
Why There Are Acne?
By Ramzaj
Why there are spots? Mechanisms of occurrence of acne, comedones and inflammations of sebaceous glands.
The single reason of occurrence of a pimple does not exist.
Two are considered as main mechanisms of evolution of acne:
1. The reinforced production of dermal grease.
2. Blockage of an ostium of a follicle.
Activity of sebaceous glands appreciablly increases during puberty. Under effect of androgens occurrence of new crates both in a sebaceous gland, and in a follicle (keratinization) is sped up. Desquamated crates inside of a follicle becomes more. Masses of keratin, the desquamated crates, mixed with a plenty of dermal grease, can block an ostium of a follicle. So there is a cork - a comedo, or acne (the mechanism of its occurrence is shown in a picture).
Evolution of a comedo (picture).
By fingers the incremented quantity of dermal grease and desquamated crates from walls of a follicle is shown.
The hair follicle is dilated and strained.
Evolution of a comedo (tissue specimen).
In an ostium of a hair follicle cork, the future comedo is shaped. Sebaceous glands are incremented, their ostiums are dilated.
Legends
1 - cork in an ostium of a hair follicle. 2 - the incremented sebaceous glands.
In the meantime, the reinforced production of dermal grease prolongs. Cork does not yield him an outcroping of a leather, and the follicle swells from grease. Sometimes there is a disrupture of a follicle.
In the blocked follicle bacteria collect. Activity of bacteria provokes an inflammation of a follicle. So there are inflammatory devices of a pimple - acne. The disrupture of a follicle result ins to extending of an inflammation around.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Why there are spots? Mechanisms of occurrence of acne, comedones and inflammations of sebaceous glands.
The single reason of occurrence of a pimple does not exist.
Two are considered as main mechanisms of evolution of acne:
1. The reinforced production of dermal grease.
2. Blockage of an ostium of a follicle.
Activity of sebaceous glands appreciablly increases during puberty. Under effect of androgens occurrence of new crates both in a sebaceous gland, and in a follicle (keratinization) is sped up. Desquamated crates inside of a follicle becomes more. Masses of keratin, the desquamated crates, mixed with a plenty of dermal grease, can block an ostium of a follicle. So there is a cork - a comedo, or acne (the mechanism of its occurrence is shown in a picture).
Evolution of a comedo (picture).By fingers the incremented quantity of dermal grease and desquamated crates from walls of a follicle is shown.
The hair follicle is dilated and strained.
Evolution of a comedo (tissue specimen).In an ostium of a hair follicle cork, the future comedo is shaped. Sebaceous glands are incremented, their ostiums are dilated.
Legends
1 - cork in an ostium of a hair follicle. 2 - the incremented sebaceous glands.
In the meantime, the reinforced production of dermal grease prolongs. Cork does not yield him an outcroping of a leather, and the follicle swells from grease. Sometimes there is a disrupture of a follicle.
In the blocked follicle bacteria collect. Activity of bacteria provokes an inflammation of a follicle. So there are inflammatory devices of a pimple - acne. The disrupture of a follicle result ins to extending of an inflammation around.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Saturday, May 5, 2007
Bacteria and acne
By Ramzaj
Bacteria - one of the main reasons of evolution of acnes. They promote an inflammation. P. acnes oozes stimulating materials and interreacts with leucocytes.
Bacteria term among the main reasons of occurrence of acnes. The role of bacteria consists in evolution of an inflammation.
Different germs can populate a leather and hair follicles. These are so-called propionibacteria: Propionibacterium acnes (P. acnes) and Propionibacterium granulosum, and also dermal staphilococcus Staphylococcus epidermidis. Except for them, on a leather and in sebaceous glands other bacteria, funguses (for example, Malassezia - a change agent of a dandruff and a seborrheal dermatitis) and ticks Demodex who can play a role in evolution of pink acnes are constantly or temporarily detected. The numbered germs do not call sicknesses of a leather in the majority of people. They are representatives of normal microflora of a leather. They are related to diseases only when for this purpose the special requirements - for example, blockage of a follicle and excessive activity of sebaceous glands are framed.
For 2-3 years before sexual maturity of the person population P. acnes on a leather is sharply incremented: from 1-10 up to millions bacteria on 1 sm2.
The quantity of bacteria on a leather and in a follicle is not related directly to gravity acne. This quantity is defined by a composition of materials on a surface of a leather, one of which bacteria use in nutriment, and others, on the contrary, interfere with their propagation. Besides propagation of bacteria is influenced with access of oxygen and acidity of a leather. P. acnes lives in a follicle below a level of a leather, not requiring in oxygen. Blockage of a follicle does not prevent, but can help manifolding P. acnes.
How bacteria call an inflammation? Ferments of bacteria of a lipase, proteinase, phosphatase and a hyalrunate-liase can split lipids, protein and other materials inside of a follicle and on a surface of a leather, to damage crates. For example, lipases split triglycerides of dermal grease ice up to the free fatty acids - nutrients. It promotes manifolding of bacteria in a follicle.
P. acnes forms the different materials serving by mediators ("messengers") of an inflammation. These materials penetrate for limits of a follicle and involve crates-leukocytes from a copulative cloth surrounding a follicle. P. acnes contacts moleculas-receptors of leucocytes then those ooze the whole gang of materials: interleukins 8 and 12, the factor of a necrosis of tumours. These materials, in turn, influence other crates that result ins to evolution of inflammatory response.
At acnes sick of heavy shapes guess the special response to bacteria - a hypersensitivity, that is some kind of an allergy.
In spite of the fact that bacteria "promote" occurrence of spots, acnes are not infectious. Participation of bacteria in evolution acne is considered secondary, but necessary for occurrence of inflammatory elements of acnes.
It explains efficiency of antibiotics and antiseptics, as one of main resorts of treatment of acnes and care of a leather, inclined to spots.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Bacteria - one of the main reasons of evolution of acnes. They promote an inflammation. P. acnes oozes stimulating materials and interreacts with leucocytes.
Bacteria term among the main reasons of occurrence of acnes. The role of bacteria consists in evolution of an inflammation.
Different germs can populate a leather and hair follicles. These are so-called propionibacteria: Propionibacterium acnes (P. acnes) and Propionibacterium granulosum, and also dermal staphilococcus Staphylococcus epidermidis. Except for them, on a leather and in sebaceous glands other bacteria, funguses (for example, Malassezia - a change agent of a dandruff and a seborrheal dermatitis) and ticks Demodex who can play a role in evolution of pink acnes are constantly or temporarily detected. The numbered germs do not call sicknesses of a leather in the majority of people. They are representatives of normal microflora of a leather. They are related to diseases only when for this purpose the special requirements - for example, blockage of a follicle and excessive activity of sebaceous glands are framed.
For 2-3 years before sexual maturity of the person population P. acnes on a leather is sharply incremented: from 1-10 up to millions bacteria on 1 sm2.
The quantity of bacteria on a leather and in a follicle is not related directly to gravity acne. This quantity is defined by a composition of materials on a surface of a leather, one of which bacteria use in nutriment, and others, on the contrary, interfere with their propagation. Besides propagation of bacteria is influenced with access of oxygen and acidity of a leather. P. acnes lives in a follicle below a level of a leather, not requiring in oxygen. Blockage of a follicle does not prevent, but can help manifolding P. acnes.
How bacteria call an inflammation? Ferments of bacteria of a lipase, proteinase, phosphatase and a hyalrunate-liase can split lipids, protein and other materials inside of a follicle and on a surface of a leather, to damage crates. For example, lipases split triglycerides of dermal grease ice up to the free fatty acids - nutrients. It promotes manifolding of bacteria in a follicle.
P. acnes forms the different materials serving by mediators ("messengers") of an inflammation. These materials penetrate for limits of a follicle and involve crates-leukocytes from a copulative cloth surrounding a follicle. P. acnes contacts moleculas-receptors of leucocytes then those ooze the whole gang of materials: interleukins 8 and 12, the factor of a necrosis of tumours. These materials, in turn, influence other crates that result ins to evolution of inflammatory response.
At acnes sick of heavy shapes guess the special response to bacteria - a hypersensitivity, that is some kind of an allergy.
In spite of the fact that bacteria "promote" occurrence of spots, acnes are not infectious. Participation of bacteria in evolution acne is considered secondary, but necessary for occurrence of inflammatory elements of acnes.
It explains efficiency of antibiotics and antiseptics, as one of main resorts of treatment of acnes and care of a leather, inclined to spots.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Friday, May 4, 2007
Acne and psychology
By Ramzaj
Ordinary acnes can reduce a self-rating and quality of life, to provoke psychologic problems. All this can be overcome, having started treatment of acnes.
Of acnes do not die. But this sickness all the same prevents to live.
One of founders of dermatology has told, that there is no sickness which has framed more than mental traumas, uncertainty, experiences and suffering, than ordinary acnes.
Very longly this fact did not note. Besides earlier acnes almost did not treat, to it did not pay attention. Acnes are at all - anything terrible, will peter.
Actually, ordinary acnes are at all « anything terrible ». They lead up some people and to depression. The main thing not only that to everyone would be desirable to be beautiful, and with acnes it does not get out.
The state of a leather can seem so to the poor, that the person with acnes avoids dialogue with other people, is ashamed to seem to them on an eye. With a problem become summer holiday and playing sports - in fact undressing, it is necessary to unclose a breast and a back.
Difficulties in dialogue with an opposite floor are especially heavy accepted. Many think, that with acnes on the face it is impossible to be attractive, somebody to like. Sometimes it is possible to hear and "comments" to address of the leather. And the some ordinary acnes frame unusual difficulties - prevent to study, get a job, promote. People lose confidence of, restrict the circle of dialogue, remain alone with the problems.
How to size up agency of acnes on quality of life and a self-rating in your event?
Having transited psychologic test APSEA or coefficient CADI.
Whether there is a medicine for these problems? Yes, there is.
Think. You not one. Acnes are at million people. They are, were or will be almost at everyone whom you know. They can be seen, for example, at kind ten Hollywood celebrities. People cope with this problem, and you it can too. Do not yield acnes of the check of your consciousness.
In any event, you always have an exit. Take, yes get rid of acnes.
If all is bad also you do not consult, but to the doctor to go be afraid, break this barrier. You do not shock the doctor - he precisely saw such events, as yours. And, most likely, both heavier. And all of them can be cured. Already some examinations have shown, that successful treatment of acnes recovers quality of life and raises a self-rating.
© Ramzaj
Buy Accutane (Isotretinoin) $2.00 per pill
Ordinary acnes can reduce a self-rating and quality of life, to provoke psychologic problems. All this can be overcome, having started treatment of acnes.
Of acnes do not die. But this sickness all the same prevents to live.
One of founders of dermatology has told, that there is no sickness which has framed more than mental traumas, uncertainty, experiences and suffering, than ordinary acnes.
Very longly this fact did not note. Besides earlier acnes almost did not treat, to it did not pay attention. Acnes are at all - anything terrible, will peter.
Actually, ordinary acnes are at all « anything terrible ». They lead up some people and to depression. The main thing not only that to everyone would be desirable to be beautiful, and with acnes it does not get out.
The state of a leather can seem so to the poor, that the person with acnes avoids dialogue with other people, is ashamed to seem to them on an eye. With a problem become summer holiday and playing sports - in fact undressing, it is necessary to unclose a breast and a back.
Difficulties in dialogue with an opposite floor are especially heavy accepted. Many think, that with acnes on the face it is impossible to be attractive, somebody to like. Sometimes it is possible to hear and "comments" to address of the leather. And the some ordinary acnes frame unusual difficulties - prevent to study, get a job, promote. People lose confidence of, restrict the circle of dialogue, remain alone with the problems.
How to size up agency of acnes on quality of life and a self-rating in your event?
Having transited psychologic test APSEA or coefficient CADI.
Whether there is a medicine for these problems? Yes, there is.
Think. You not one. Acnes are at million people. They are, were or will be almost at everyone whom you know. They can be seen, for example, at kind ten Hollywood celebrities. People cope with this problem, and you it can too. Do not yield acnes of the check of your consciousness.
In any event, you always have an exit. Take, yes get rid of acnes.
If all is bad also you do not consult, but to the doctor to go be afraid, break this barrier. You do not shock the doctor - he precisely saw such events, as yours. And, most likely, both heavier. And all of them can be cured. Already some examinations have shown, that successful treatment of acnes recovers quality of life and raises a self-rating.
© Ramzaj
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